BrianD
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Everything posted by BrianD
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Terri has an excellent point here, making the designation of restricted access areas well-known during "disaster-preparedness" training of staff is a very good idea...(especially in our swampy climate and a power-outage occurs....the blood bank keeps AC)
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i would strongly recommend the mother's ab screen and the infant's tranfusion history be re-evaluated.
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lol, i think that's a common "condition" amongst blood bankers, Malcolm. i'll introduce some very highly technical jargon from my "practice:" The Heap: this is the term for my desk, you can't actually see it beneath the papers and post-it notes. The Pile: this is the term for the collection of books needing to be read. There is a Queen Anne style chair under there.....somewhere.
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i keep putting the capability to remove the lock out on electronic crossmatches for the patient once the passively acquired anti D (RhIg) titers down and the patient reverts back to a negative ab screen on my "quarterly wish list" but apparently revising the truth-tables is akin to redacting the Bible 'round here. one day.........
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i'd expect there is, M. but *********************************************************************************** ***********************************************. :explosion oops, my frustrations with the IS types are showing.:pcproblem
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our hospital has been using SWANK for about 2 years now and so far so good tho' i've not been as impressed with it's course-authoring component. when i was teaching, i used Blackboard and really liked it. tho' unless your institution is doing a lot of teaching, Blackboard might be a bit too expensive.
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our lab currently reflexively runs a complete ID panel tho' given our saturation (6 hospitals, and 20-somethin' clinics) we know who has been dosed with RhIg most of the time. the current argument is regarding reporting a positive ab screen. the ab screen in our procedure manual is "for the detection of unexpected antibodies directed against red cell antigens." those of us who are older would prefer to report the ab screen as negative since we expect the passively acquired ant-D to be there since we put it there in the first place. the younger folks just roll their eyes at us older folks as being unnecessarily nit-picky but complain loudest about the patient being forever disqualified from eligibility for electronic crossmatches and must have AHGXMs since the patient has a positive ab screen in the permanent record.
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lol, i'd support this.......i'd love to finish my years of practice without ever dealing with anti-Kell HDFN ever again. i'd also encourage OB/Gyn clinics to get educated about anti Lu(a) and to stop scaring the crap out of expectant mothers with it.
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Merry Christmas, everybody.
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you're on the right track! this particular haplotype cannot align correctly with comparable haplotypes because it is shortened due to a deletion of a segment of the Class III MHC cluster which removes the C4A gene which carries the Rodgers antigen determinant so it would be unexpected for Rodgers antigen to be expressed from this haplotype
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here's an easy-peasy question to keep the ball rolling: what is the reason the HLA haplotype: A1,B8,DRB1*0301 is believed to be resistant to recombination AND which blood group antigen cannot be expressed from this particular haplotype.
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"hen-pecked??" is that the 'politically-correct' phrase the kids are using these days?? LOL
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i think a sense of humor is vital and think an addition that would benefit any clinical training curriculum is something geared toward the development of good humor. i loved how my nurse was baffled when i was checked in for radiation therapy and when completing the questionnaire, my response to "why are you here, today?" was "my hedgehog has gone wonky."
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if i'm understanding your question correctly, EDTA plasma is fine for manual testing.....as Malcolm Needs pointed out in his response, the need for active complement in Ab detection is a very very rare occurrence. i just have a personal preference for having access to a paired serum sample for manual antibody i.d. work.....usually, i can get plenty of plasma/serum....red cells are what i never seem to have enough
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'tis a pity, that. LOL i think blood group serologists want to have as much fun naming things as the fruit fly geneticists did naming the genes they were studying : teashirt, tinman( embryos lack a heart), dreadlocks, cheapdate, lush,Van Gogh (wings have swirly hair patterns), grim reaper, Cleopatra (mutant protein interacts with a protein called Asp and the interaction is lethal), Hamlet(controls the development of the IIb cells in the embryo), gypsy, decapentaplegic, mothers-against-decapentaplegic, daughters-against-decapentaplegic, frizzled, frazzled, Seven-less, Bride-of-Sevenless. it goes on and on. unfortunately, similar mutations have been found in in humans and are associated with awful diseases.
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Donor on treatment of antidepressant drugs
BrianD replied to Desoki's topic in Donor Services and Donor Recruitment
i've often wondered about this situation, especially given some of the psychoactives are being found to be teratogenic. still, i believe that the DHQ eliminates most of the concern as Deb has pointed out since if there is a suggestion of a lack of reliability in the self-reported medical history then the donor is deferred. -
EDTA plasma is what we use for most things, tho if i have to do a manual ab id panel i like to use serum.....Kidd abs seem to come up better in serum but the automated plate assays we use are sensitive enough that we can pick them up in EDTA plasma 95% of the time.
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prolly because ZENA and CRAM were already taken LOL
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LOL, devilish question aside, i don't think you'll be called "The Tooting Meadow Torquemada" anytime soon. This is a good one tho'. i'm thinking i might use this question on my assessment test for the new clinical students......a well constructed test MUST have a question that only 1 in 100 students would have a chance of answering. heh heh. tho' if my last crop of them are any indication this question will fall into the 1 in 10,000 interval.
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:chainsaw::chainsaw:I am slain!! I’m aware of no new Kell system antigens after KUCI, KANT, and KASH. I resorted to some googly-ness (is this cheating?!?!) and all I found was an interesting if a bit odd paper detailing the use of immunocytochemistries to elucidate the synthesis and distribution of l- and k- carrageenans in seaweeds. The authors called their anti-k-carrageenan ab “anti-K.” :confused::cries::confused: this question after bombing my Donor Center Operations exam. oy. :shakefist:shakefist :surrender:surrender:surrender:surrender:surrender i will now spend the rest of the day contemplating running away and becoming a librarian.
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one of our attendings in the ER has a unique sense of humor, and when the new admissions clerk asked for an admitting diagnosis for a GSW he told her "acute lead poisoning".
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yes!!! Kell. the other polypeptide it is associated with is Kx (Xk) to which it is connected by a single disulfide linkage. next question goes to goodchild!
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here's a question: Which blood group system is unique in that the antigens are located on a Type II membrane glycoprotein: it traverses the cell membrane only once and has a large extracellular domain having 15 cysteine residues producing elaborate conformational folding through disulfide bonding. In addition to being present on erythroid cells, this system of blood group antigens can also be detected in myeloid progenitor cells, testis, lymphoid tissues, and in skeletal muscle.
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and the next question is??? c'mon someone, i started SBB skewl and i'm relying on y'all to help me be a real smarty pants:imslow: i think i'll end up in the nut-house or the jail-house before this is over.....taking on this stuff and just "inheriting" ANAs and the first read-out on anaerobes. :cries::cries::cries::cries::cries::cries::cries::cries::cries:
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have a great time and do avoid the cider. (the lab i worked in had an "incident" precipitated by an excess of WoodChuck Green Apple Cider and i won't go near the stuff!).