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Can I just remind everyone that whatever you do, you will NEVER provide anti-D to everyone who needs it and avoid giving to everyone who doesn't - at least not until someone comes up with a foolproof, cheap and quick way of genotyping. Some partial Ds (who should receive anti-D) will be missed because they react exactly like normal D+, unless you are lucky enough to have a clone that misses the one epitope that they are lacking too. Not all partials have decreased amounts of D antigen, and not all weak Ds can be sensitised to the D antigen. This is 'grey areas' in spades. The word 'simple' just does not exist when talking about Rh.

Totally agree. We encountered same with ProVue....we tried running IS on buffered gel card and did not detect ABO incompatibility on one occasion!! Try fighting with regulating bodies .....CLIA interpretation with Ortho letter......Most sites gets cited if you are not shaking tubes for IS when using gel methods......If your computer system is able to detect ABO incompatibility at the unit selection, is far better than doing IS by tube with Gel crossmatch.................but no one wants to listen and the reason being when people get cited they blindly do IS instead of justifying why computer is better than shaking tube for IS......

This may be a silly question...but are you sure he understands the difference between pheresis and random donor? The vast majority of our physicians still order 5-6 packs of platelets even though we have only carried pheresis for 10+ years.